Friday, May 10, 2019
Anesthetic Management of the Patient with Aortic Stenosis Essay
Anesthetic Management of the longanimous with Aortic Stenosis - Essay ExampleSurgery is indicated in many of the cases in order to secure or replace the diseased valve. Therefore, it is the purpose of this paper to investigate the current anesthetic management of this disease, from surgical to pos put acrosserative care. This paper will describe the basic anatomy and physiology of the affection, in particular, the aortal valve. The pathophysiological changes in the aortic valve and the heart will also be discussed.The heart is a hollow muscular organ that is or so pyramid-shaped and lies within the inferior mediastinum (3). It is covered by a fibroserous sac called the pericardium, within which pericardial fluid fills the plaza between the serous and fibrous layers. The pericardial fluid acts as a lubricant to prevent corrasion during the contraction of the heart. The pericardium is not very distensible, and therefore it strongly resists a large, rapid increase in cardiac size . Hence, the pericardium also prevents sudden overdistention of the chambers of the heart (4). The heart is divided by a vertical septa into quaternion chambers the right and left atria, which occupy the upper chambers and the right and left ventricles, which occupy the tear down chambers. ... Although most myocardial cells are able to perform both these functions, the vast majority is predominantly contracted cells (myocytes) and a small number are specifically designed as electrical cells. The latter, collectively cognise as the conducting system of the heart, are not nervous tissue but modified myocytes lacking in myofibril components. They have the ability to generate electrical impulses which are then conducted to the myocytes, leading to contraction by a process known as excitation-contraction coupling (5). In order to produce a synchronised contraction, there must be a normal spread of electrical activity within the heart. depolarization is initiated in the sinoatrial no de (SA) node hence it is known as the pacemaker of the heart. It sets the contractions of the heart at a rate of 60-100 beats per minute. From the SA node, the electrical impulse travels to the atrioventricular (AV) node and then propagates through the Bundle of His. From the top of the septum, the wave of depolarization spreads in the rapidly conducting Purkinje fibers to all parts of the ventricles (6).Between the atria, ventricles and great blood vessels arising from the heart (aorta and pulmonary artery) are valves, which are made up of cusps or leaflets to prevent backflow of blood, ensuring a one-way conduit of blood through the chambers and to the pulmonary and systemic circulation. There are four valves ii crescent-shaped valves (aortic and pulmonary valves) and two atrioventricular valves (tricuspid and mitral valves). All of these valves have 3 cusps except the mitral valve which has two cusps. Unoxygenated blood from the superior and inferior vena cava and coronary
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